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The description of highly exposed individuals who remain seronegative HESN despite repeated exposure to human immunodeficiency virus HIV -1 has heightened interest in identifying potential mechanisms of HIV-1 resistance.

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Together with data illustrating increased activation [ 10 , 20 , 91 ] and function [ 6 , 19 ] of NK cells in HESNs, these results suggest that NK cells fit the model of a candidate cell type whose retained function and heightened activation status may contribute to both control over HIV-1 replication and resistance to HIV-1 in HESN subjects. Defensins [ 79 — 81 ].

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Escort 388 no The case for the anti-viral capacity of NK subsets during infection is suggested by its loss of function in chronic infection.

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